Ventricular Septal Defect

Below you will find more information about Ventricular Septal Defect from Medigest. If you believe that you are suffering from any of the symptoms of Ventricular Septal Defect it is important that you obtain an accurate diagnosis from a medical professional to ensure that you obtain the correct medication or treatment for your condition. There are medical conditions that carry similar symptoms associated with Ventricular Septal Defect and therefore the information provided by Medigest is offered as a guideline only and should never be used in preference to seeking professional medical advice. The information relating to Ventricular Septal Defect comes from a third party source and Medigest will not be held liable for any inaccuracies relating to the information shown.

Definition

Ventricular septal defect or VSD for brevity is a defect in the wall dividing the left and right ventricles of the heart called the ventricular septum which consists of an inferior muscular and superior membranous portion which is close to the atrioventricular node, It is the most common congenital heart defects in adults and older children (Hoffman JI, Kaplan S. (2002) The Incidence of congenital heart disease, p 39).

Diagnosis

Ventricular septal defect or VSD is usually detected by cardiac auscultation which is generally considered sufficient for detecting a significant VSD.

Treatment

Treatment depends on the extent of the opening due to the VSD. Small congenital VSD often close on its own as the heart grows. On the other hand in cases requiring surgical intervention, a heart-lung machine is required and a median sternotomy is performed. Percutaneous endovascular procedure which is less invasive is also used. Administration of Digoxin and Frusemide are also used to treat VSD.

Symptoms and Signs

Most infants with VSD are asymptomatic until pulmonary vascular resistance begins to fall at approximately 4- 25 weeks of age. At this point, the infant normally breathes more rapidly and show signs of cyanosis, feeds poorly and is diaphoretic (Porth, Carol (2006) Essential of Pathophysiology: Concepts of Altered Health States, Lippincott Williams and Wilkins, p. 413).

Causes

It is often attributed to the higher systolic pressure of the left ventricle which causes leakage of blood into the right ventricle elevating its pressure and volume which in turn cause pulmonary hypertension, an associated symptom of the VSD.

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